Antifreeze/coolant Poisoning and Prevention

        Each year more than ten thousand dogs and cats are accidentally poisoned with automotive antifreeze/coolant. One step owners can take to prevent a potential tragedy is to use the less-toxic propylene-glycol-based antifreeze in the car. Propylene glycol  - although not entirely nontoxic - is considerably less toxic than ethylene glycol. Although dogs and cats account for most cases, ethylene glycol is toxic to all animals - including people. It is one of the top ten causes of poisoning in dogs and cats in the United States. The toxic ingredient is its major component and makes up 95% of the product. It only takes a small sip of antifreeze to poison your pet; a few ounces are lethal. Cats are approximately four times as sensitive to the poison as dogs. For a medium sized dog, ingestion of about 2 ounces (3-4 tablespoons) is toxic.  For a cat little as 1/4 of an ounce (1-2 teaspoons) can be lethal.  The problem is compounded by the fact that this stuff has a sweet taste and when animals find it, they enjoy the taste.

        Antifreeze poisoning commonly occurs in spring and fall when car owners replace the old antifreeze with fresh antifreeze in their car radiators. However, poisoning can happen anytime, particularly when a car boils over or a hose leaks. This poisoning happens often to animals who are allowed to roam freely in their neighborhoods, but another high risk group is dogs who are confined in garages and who may not always be provided with adequate fresh drinking water. Additionally these dogs may gain access to improperly or inadequately stored antifreeze or lick spilled or leaked antifreeze off the garage floor. If it is necessary to confine your pet(s) to your garage, make sure antifreeze containers are well secured and your animal has plenty of fresh water. Spills can be cleaned up with kitty litter as an absorbent, being sure that you remove and dispose of the litter quickly and completely, then a thorough flushing of the affected area.  Antifreeze is biodegradable but it may take several months.

        Ethylene glycol is also found in brake fluid, liquid rust inhibitors, hydraulic fluids and solar collectors. Another source is ethelyne glycol 2% in the liquid placed inside of decorative “snow scene” glass globes which one inverts to see the flocculent snow fall. Be sure to protect not only your pets but your children from breaking and possibly ingesting material from these decorative items.

        Ethylene glycol has an immediate and a long-term affect on the body. It is rapidly absorbed and metabolized once it has been consumed. Peak blood levels occur within three hours of ingestion. Within thirty minutes after drinking it, your pet will become ataxic, or drunken, in appearance. This phase continues for up to six hours. The animal appears to recover but by then the liver will have begun metabolizing the ethylene glycol into to glyoxcylic acid, formic acid, and oxalate which destroy renal tubular cells in the kidneys causing kidney failure with the resulting uremia. These compounds also seriously damage the central nervous system. There is no treatment that will reverse this damage.

        Dogs and cats can only be cured when the poisoning is detected before extensive kidney damage has occurred. Diagnosis is not difficult when an owner presents a pet that is staggering and drunken in appearance and has seen the animal drink the antifreeze/poison. It is much more difficult when the ethylene glycol first reaches the liver because early in this stage the pet will appear healthy while later in this stage symptoms are multi-systemic and nonspecific. These signs can be confused with other diseases such as pancreatitis, acute gastroenteritis, diabetes or other forms of kidney disease. By the time ethylene glycol metabolites have attacked the kidneys it is too late for a cure. By this time the animal is very sick from uremia and acidic blood (acidosis). In unfortunate animals that die, it is the six-sided or Maltese-cross shaped crystals of calcium oxalate within kidney tubules that allow pathologists to make the diagnosis in a post-mortem exam. Sometimes the urine of affected pets will glow when exposed to a woods or ultraviolet lamp.

        The amount of ethylene glycol the animal consumed is very important in determining the success of treatment. Animals do not respond favorably to any treatment when they have ingested too much. Treatment is based on decreasing the absorption of ethylene glycol from the stomach and intestine and increasing its excretion through the kidneys. Preventing metabolism of ethylene glycol to glycolic acid and calcium oxalate and correcting acidosis of the blood is also very important. The first step is to administer apomorphine or peroxide solution to get the pet to vomit up any poison remaining in the stomach. Next the animal is given water between medications in order wash out the stomach thoroughly (gastric lavage). Finally activated charcoal is administered to bind with any poison that is left. Large amounts of intravenous fluids are given simultaneously to increase urine production and excrete as much ethylene glycol as possible.  Treatment was discovered accidentally when a group of teenagers consumed antifreeze accidentally during a party. It was found that the teenagers who were the drunkest suffered the least side effects from the poison. That information is utilized today in treating pets giving them grain alcohol -To be effective, this must begin within 6 hours after ingestion.

        If you suspect your pet had ingested antifreeze DO NOT hesitate to get them to your veterinarian immediately. We have shared what is involved in trying to treat this poisoning to try to impress upon you how serious this condition is. The only sure treatment is to prevent antifreeze poisoning before it happens. Be sure that your pet is protected from contact with antifreeze and coolants - given the chance they will lick it due to the sweet taste. It's up to you to keep them safe from it.



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Last updated 1/2005                     © Inclusive from 1999 NACR